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Myocardial scar and LVEF are both risk markers for all-cause and cardiac death in patients with nonischemic cardiomyopathy. However, whereas myocardial scar has strong and . The clinical profile and arrhythmic outcome of competitive athletes with isolated nonischemic left ventricular (LV) scar as evidenced by contrast-enhanced cardiac magnetic .

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Myocardial scar and LVEF are both risk markers for all-cause and cardiac death in patients with nonischemic cardiomyopathy. However, whereas myocardial scar has strong and incremental prognostic value for SCD risk stratification, LVEF has no incremental prognostic value over clinical measures.

The clinical profile and arrhythmic outcome of competitive athletes with isolated nonischemic left ventricular (LV) scar as evidenced by contrast-enhanced cardiac magnetic resonance remain to be elucidated.

The identification of a nonischemic left ventricular (LV) scar represents a significant challenge for cardiologists due to the need for differential diagnosis among a wide range of diseases, with major diagnostic, therapeutic, and prognostic implications for patients and their families.

Left ventricular (LV) scar on late gadolinium enhancement (LGE) cardiac magnetic resonance has been correlated with life-threatening arrhythmic events in patients with apparently idiopathic ventricular arrhythmias (VAs).

Patients with chronic Chagas cardiomyopathy (CCC) have pronounced myocardial fibrosis, which may predispose to sudden cardiac death, despite well-preserved global left ventricular (LV) systolic function. Cardiac magnetic resonance can assess myocardial fibrosis by late gadolinium enhancement (LGE) sequences.Introduction: The extent of left ventricular (LV) scar, characterized by late gadolinium enhancement cardiac MRI (LGE-CMR), has been shown to predict the occurrence of ventricular arrhythmias in implantable cardioverter defibrillator (ICD) recipients.

Ventricular scars are a major cause of ventricular tachycardia in many forms of heart disease. Methods to identify and characterize scar hold promise for identifying patients at risk.

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The amount of LV scar might modulate the impact of FMR on the clinical course, and especially in patients within the extreme ranges of scar burden (no/limited and extensive scar), a careful assessment is needed to consider TMVI in the presence of significant FMR. A left ventricular (LV) scar is defined as “non-ischemic” when it affects the subepicardial/midmyocardial layer of the LV wall, contrasting with the ischemic LGE that is subendocardial/transmural, and “isolated” when no history or signs of . In patients with ischemic cardiomyopathy, the LV scar burden correlated negatively with the increase in LVEF after CRT and was associated with worse outcome. 10 Therefore, our study may indicate that in patients with scar progression, CRT is unlikely to result in reverse cardiac remodeling. Myocardial scar and LVEF are both risk markers for all-cause and cardiac death in patients with nonischemic cardiomyopathy. However, whereas myocardial scar has strong and incremental prognostic value for SCD risk stratification, LVEF has no incremental prognostic value over clinical measures.

The clinical profile and arrhythmic outcome of competitive athletes with isolated nonischemic left ventricular (LV) scar as evidenced by contrast-enhanced cardiac magnetic resonance remain to be elucidated.The identification of a nonischemic left ventricular (LV) scar represents a significant challenge for cardiologists due to the need for differential diagnosis among a wide range of diseases, with major diagnostic, therapeutic, and prognostic implications for patients and their families. Left ventricular (LV) scar on late gadolinium enhancement (LGE) cardiac magnetic resonance has been correlated with life-threatening arrhythmic events in patients with apparently idiopathic ventricular arrhythmias (VAs).

Patients with chronic Chagas cardiomyopathy (CCC) have pronounced myocardial fibrosis, which may predispose to sudden cardiac death, despite well-preserved global left ventricular (LV) systolic function. Cardiac magnetic resonance can assess myocardial fibrosis by late gadolinium enhancement (LGE) sequences.

Introduction: The extent of left ventricular (LV) scar, characterized by late gadolinium enhancement cardiac MRI (LGE-CMR), has been shown to predict the occurrence of ventricular arrhythmias in implantable cardioverter defibrillator (ICD) recipients.

Ventricular scars are a major cause of ventricular tachycardia in many forms of heart disease. Methods to identify and characterize scar hold promise for identifying patients at risk. The amount of LV scar might modulate the impact of FMR on the clinical course, and especially in patients within the extreme ranges of scar burden (no/limited and extensive scar), a careful assessment is needed to consider TMVI in the presence of significant FMR. A left ventricular (LV) scar is defined as “non-ischemic” when it affects the subepicardial/midmyocardial layer of the LV wall, contrasting with the ischemic LGE that is subendocardial/transmural, and “isolated” when no history or signs of .

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lv scar|1.5 luv scars ko 1600

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